From f27676b31611a0577effcbf7e31c523a36fc42ed Mon Sep 17 00:00:00 2001 From: Dotty Gibney Date: Wed, 30 Jul 2025 10:02:31 +0000 Subject: [PATCH] Update 'What's Biogenix Relief Glycogen Support Extreme?' --- What%27s-Biogenix-Relief-Glycogen-Support-Extreme%3F.md | 1 + 1 file changed, 1 insertion(+) create mode 100644 What%27s-Biogenix-Relief-Glycogen-Support-Extreme%3F.md diff --git a/What%27s-Biogenix-Relief-Glycogen-Support-Extreme%3F.md b/What%27s-Biogenix-Relief-Glycogen-Support-Extreme%3F.md new file mode 100644 index 0000000..0117bb1 --- /dev/null +++ b/What%27s-Biogenix-Relief-Glycogen-Support-Extreme%3F.md @@ -0,0 +1 @@ +
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In nerves uncovered to one hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose further energy substrate is required when the tissue vitality demand will increase. Such data are necessary as they show that glycogen has an essential position underneath physiological circumstances. The truth that glycogen supported CAP conduction in the absence of exogenously applied glucose probably has no value in ascribing a role to glycogen, since aglycaemia is an synthetic experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that did not exist previous to 1922 (Frier et al., 2014), thus cannot be glycogen’s foremost role. However supplying lactate to axons when tissue vitality demand will increase implies a novel function for brain glycogen and suggests it acts as an vitality buffer somewhat than an vitality reserve, whose position is to not single-handedly support conduction, however to act in concert with ambient ranges of normoglycemic glucose.
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